Grupo 2
Fisiopatología Cardiovascular
- Director/a
- Victoria Cachofeiro Ramos, Ernesto Martinez Martinez
- Tipo de grupo
- CONSOLIDADO
- Líneas de investigación
-
Axis 1: Obesity-associated cardiovascular and metabolic damage L1: Role of metabolic substrate utilization switching in obesity-associated cardiometabolic damage
L2: Role of mitochondrial dysfunction in obesity-associated cardiometabolic damage
L3: Role of endoplasmic reticulum stress in obesity-associated cardiometabolic damage
L4: Role of autophagy in obesity-associated cardiometabolic damage
L5: Impact of mitochondrial transplantation on cardiac function and structure
L6: Impact of "Mitochondria-like mitochondrialuterions" on obesity-associated cardiac damage
Axis 2: Mechanisms involved in cardiovascular damage associated with myocardial ischaemia.
L1: Role of changing metabolic substrate use in ischaemia-associated cardiometabolic damage
L2: Impact of obesity on cardiac alterations associated with myocardial ischaemia.
L3: Role of changing metabolic substrate use in cardiometabolic damage associated with myocardial ischaemia.
L4: Role of mitochondrial dysfunction in cardiometabolic damage associated with myocardial ischaemia
L5: Role of endoplasmic reticulum stress in cardiometabolic damage associated with myocardial ischaemia
L6: Role of autophagy in cardiometabolic damage associated with myocardial ischaemia
L7: Impact of mitochondrial transplantation on cardiac function and structure
L8: Impact of "Mitochondria-like mitochondrialuterions" on cardiometabolic damage associated with myocardial ischaemia
Axis 3: Mechanisms involved in damage associated with cardiorenal syndrome.
L1: Role of mitochondrial dysfunction in renal damage associated with cardiorenal syndrome
L2: Role of endoplasmic reticulum stress in renal damage associated with cardiorenal syndrome
L3: Impact of mitochondrial transplantation on renal damage associated with cardiorenal syndrome