Investigación / Grupos de investigación

Grupo  2

Fisiopatología Cardiovascular

Director/a
Victoria Cachofeiro Ramos, Ernesto Martinez Martinez
Tipo de grupo
CONSOLIDADO
Líneas de investigación
Axis 1: Obesity-associated cardiovascular and metabolic damage

 

L1: Role of metabolic substrate utilization switching in obesity-associated cardiometabolic damage

L2: Role of mitochondrial dysfunction in obesity-associated cardiometabolic damage

L3: Role of endoplasmic reticulum stress in obesity-associated cardiometabolic damage

L4: Role of autophagy in obesity-associated cardiometabolic damage

L5: Impact of mitochondrial transplantation on cardiac function and structure

L6: Impact of "Mitochondria-like mitochondrialuterions" on obesity-associated cardiac damage

Axis 2: Mechanisms involved in cardiovascular damage associated with myocardial ischaemia.

L1: Role of changing metabolic substrate use in ischaemia-associated cardiometabolic damage

L2: Impact of obesity on cardiac alterations associated with myocardial ischaemia.

L3: Role of changing metabolic substrate use in cardiometabolic damage associated with myocardial ischaemia.

L4: Role of mitochondrial dysfunction in cardiometabolic damage associated with myocardial ischaemia

L5: Role of endoplasmic reticulum stress in cardiometabolic damage associated with myocardial ischaemia

L6: Role of autophagy in cardiometabolic damage associated with myocardial ischaemia

L7: Impact of mitochondrial transplantation on cardiac function and structure

L8: Impact of "Mitochondria-like mitochondrialuterions" on cardiometabolic damage associated with myocardial ischaemia

Axis 3: Mechanisms involved in damage associated with cardiorenal syndrome.

L1: Role of mitochondrial dysfunction in renal damage associated with cardiorenal syndrome

L2: Role of endoplasmic reticulum stress in renal damage associated with cardiorenal syndrome

L3: Impact of mitochondrial transplantation on renal damage associated with cardiorenal syndrome